Alzheimer's patients have Type 2 diabetes, which is associated with increased Advanced Glycation End Products (AGEs) and increased expression of the AGED receptor.

Advanced glycation end products (AGEs) are lipid or protein materials that become exceptionally glycated because of contact with sugars. They are a bio-marker embroiled in maturing and the turn of events, or declining, of numerous degenerative sicknesses, like diabetes, atherosclerosis, persistent kidney infection, and Alzheimer’s illness

They assume a significant part in the turn of events and maturing of both solid and unfortunate cells, just as in the event and movement of a few degenerative illnesses. These incorporate cirrhosis of the liver, greasy liver, and various sclerosis. They additionally cause the obliteration of the red platelet (RBC), the development of advanced glycation end products or AME (products of glycation) inside the red platelets and the untimely maturing of tissues. The greater part of these proteins are shaky and changes their capacity when presented to shifting sugars. This interaction can be set off by various natural variables, including corpulence, persistent liquor misuse, hormonal changes, insulin obstruction, tobacco use, calming drugs, meds utilized in the therapy of malignancy, and ecological poisons.

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